Effects of Alcohol Consumption to the Cognitive Function of a Person

Excessive intoxicant intake has a large impact on the cognitive subprogram of a psyche. It leads to a person to have a poor cognitive tend. Which can be stirred the persons perception, attention, memory, motor skills, language, visual and spacial processing. To some extent, study indicates that high train of impulsivity in intoxicant purposes of a person has an stamp on the facade lobe related to their behavioral problem (Lyvers et. al. ). Furthermore, inebriant intoxication causes disruption of anterior cortical functioning and in that locationby impairs executive cognitive performance.As executive cognitive ability is concerned this is the prefrontal cortex, which is responsible for problem solving, cognitive flexibility, planning, organization, pilfer reasoning and social conduct. A person who is in inebriated post is more likely to do risky, impulsive behavior such as unprotected sex, violence and drunk driving. In some situation, heartbreaking alcoholics gets complication, grievous essential cerebral declension it is the common complication occurring in more or less 10 percent of patient (Horvath 1975).Lishman 1981 American Psychiatric Association 1987, the assorted signs of unsafe top dog dysfunction that persist after cessation of alcohol consumption have been conceptualize in terms of 2 organic mental disorders alcohol amnesic disorder (memory disorder) and dementia associated with alcoholism. Alcohol amnestic disorder, commonly called Korsakoffs psychosis or Wernicke- Korsakoff syndrome, it is characterized by short term memory, impairments and behavioral changes that occur without clouding of consciousness or general hurt of knowing abilities.Dementia associated with alcoholism consist of global loss of intellectual abilities with an impairment in memory function together disturbance(s) of abstract thinking, judgment, other higher cortical function or personality change without a clouding of consciousness. These two menta l disorders argon also cause by a severe deficiency ofThiamine(vitamin B1) and argon often precipitated by a fulminant inflow of glucose. A human activity of things have been shown to lead to a severe enough aneurin deficiency to trigger wet learning ability.Wet instinct or Wernicke-Korsakoff Syndromehas a sudden onsetit is not something which happens gradually over time. The origin stage of wet mavin is called Wernickes encephalopathy. When in that location is a sudden influx of glucose in a promontory which is deprived of thiamine the brain cells jump to die. This is because the chemical reactions which supply these brain cells with energy for life use thiamine to turn glucose into energy in a chemical process called the Krebs cycle.When there is an influx of glucose and no thiamine to help metabolize it, these brain cells blow up out like a car engine running on high octane gasoline at high speed with no oil. The brain cells which die first are the ones which require the most thiamine to function. These brain cells are located around the middle of the brain and are the brain cells which are associated with memory and muscular movement. The brain cells of the cerebellum, which controls balance, are also affected.Korsakoffs syndrome includes loss of past memories, inability to learn new things, confabulation (remembering things which never happened), wish of coordination and unsteady gait, and in severe cases dementia. These are the possible reaction to a person who takes excessive alcohol consumptions. More evident in explaining the effect of excessive intake of alcohol in a persons cognitive function is that he/she will experienced less effective at certain learning tests and visual- spatial integration. Likewise it cause wrong aging (Tarter and Edwards, 1986) and it is shut away under active investigation.In addition, it has long been an established particular that actively drinking, alcohol dependent subjects have smaller brain volumes th an formula control subjects who do not drink alcohol. Thus, early researchers assumed that this was because alcohol killed the brain cells of alcohol dependent subjects, unless other research disapproves this venture but it could be partially true unless there has been brain prostitute as a result ofliver failureorthiamine deficiency, the majority of brain cells of backbreaking drinkers are intact even though the brain has shrunk.Meanwhile, in some study stated that alcohol consumption in a moderate mode has an effect to help a person to fall down the incidence of having diabetes. Likewise, it can help to preserve brain vasculature prevents sub-clinical strokes and could result in better cognition function. But drinking in addition much of alcohol leads to have poor cognitive function that causes to a person to have brain damage. Some research shows that alcohol adversely affects the brain.When health professionals encounter patients who are having cognitive difficulties, suc h as impaired memory or reasoning ability, alcohol use may be the cause of the problem. The human brain consists of face cloth cells and gray cells. The gray cells are responsible for thinking and feeling and decisionsthey correspond to the Central Processing unit (CPU) of the computer. The white cells are like the cables of the computer which connect the keyboard and the monitor to the CPU.Jensen and Pakkenberg (1993) did brain cell counts which compared the number of cells in the brains of heavy drinkers with those of non-drinkers. They found out that the number of gray cells was the same in both the heavy drinkers and the non-drinkers. However, there were fewer white brain cells in the brains of the drinkers which imply that alcohol kills a white brain cell that is responsible to the connection to pass the message or transaction of the brain to connect with the feeling and decision making of a person.Hence, excessive alcohol intake disrupts the connection or breaks the normal function of the brain to react and to have a better cognitive function. Moreover, George Fein (2009) detect that there was one part of the brain in the parietal lobewhich is associated with spatial processingwhere alcohol kills gray cells. Fein claims that this explains why even after alcohol dependent subjects regain use of all their other cognitive functions they still seem to have difficulties with spatial processing.Parker et al. (1983) stated that there is significant decrease in test performance have been found for mountain whose self-reported alcohol consumption was in the range of what was considered social drinking. They found it out when they conducted a certain neuropsychological tests, the results of one general population study (Bergman et al. 1983). Those people were not clinically impaired they only exhibited certain performance deficits that gibe with alcohol consumption.Parsons (1986) concluded that data on the relationship of cognitive impairment to amount of al cohol consumed by social drinkers are inconclusive but has the chance to have similar correlation. There is some evidence that both the amount of brain shrinkage and the amount of cognitive deficit are dependent on the quantity of alcohol consumed and the number of years of heavy drinking. Thus, it only proves that there is an adversely effect on the cognitive function of a person in drinking too much of it. Hence, there is no good effect on the brain or form function of a person. (copyright of ISLA BONITAS 2012)